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HealthConsider > Blog > Healthcare > Japanese Encephalitis — Clinical Types, Diagnosis, Treatment, and Prevention
Healthcare

Japanese Encephalitis — Clinical Types, Diagnosis, Treatment, and Prevention

Last updated: August 11, 2025 11:47 pm
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Japanese Encephalitis (JE)

Japanese encephalitis is a mosquito‑borne flavivirus infection. Most infections are asymptomatic; a small proportion progress to encephalitis with high fever, altered mental status, and seizures. Early recognition and supportive critical care improve outcomes.

Contents
  • Japanese Encephalitis (JE)
    • Clinical Severity Types
    • Laboratory and Diagnostics
    • Treatment (Supportive; No Proven Antiviral)
    • Prognosis
    • Prevention
    • Red Flags — Seek Urgent Care

Clinical Severity Types

  • Mild: alert or drowsy; fever 38–39°C; possible meningeal signs; CSF pleocytosis. Symptoms usually resolve within 7–10 days and can resemble viral meningitis outside outbreak seasons.
  • Moderate: lethargy or brief coma; fever 39–40°C for 4–5 days; brief seizures; clear meningeal signs; possible cranial nerve palsies or limb weakness. Most recover within ~2 weeks.
  • Severe: coma; persistent fever >40°C; frequent seizures; pyramidal signs; brainstem involvement may cause respiratory distress and herniation risk. Recovery takes 2–4 weeks if cerebral edema is survived; neuropsychiatric symptoms may persist in recovery.
  • Extremely severe (≈5%): abrupt onset; temperature ≥40–41°C; refractory seizures, deep coma, massive cerebral edema; death may occur within 1–2 days from respiratory failure or herniation.
  • Atypical presentations: brainstem encephalitis, meningoencephalitis, myelitis, movement disorders.

Laboratory and Diagnostics

  • CBC: leukocytosis (often 10–20 × 10^9/L; higher possible).
  • CSF: opening pressure elevated; cells 50–500/µL, neutrophils early shifting to lymphocytes by day 4–5; protein normal to mildly high; glucose typically normal.
  • Confirmatory tests: JEV‑specific IgM (MAC‑ELISA) in CSF and/or serum (usually detectable by day 3–8 of illness); PCR/NAAT useful early. MRI may show thalamic/basal ganglia/brainstem changes.
  • Differential: HSV encephalitis, enteroviral meningoencephalitis, cerebral malaria (travelers), bacterial meningitis, heatstroke in summer.

Treatment (Supportive; No Proven Antiviral)

  • Stabilize ABCs; admit moderate/severe cases; ICU if encephalopathy, seizures, or respiratory compromise.
  • Seizure control: benzodiazepines → second‑line antiepileptics.
  • Manage fever and fluids; maintain electrolytes and nutrition.
  • Raised ICP: head‑of‑bed elevation, cautious fluids; consider hyperosmolar therapy with specialist input and neuroimaging.
  • Empiric coverage: initiate IV acyclovir (for HSV) and appropriate antibiotics if bacterial meningitis can’t be excluded; de‑escalate when ruled out.
  • Corticosteroids/antivirals are not routine for JE.

Prognosis

  • Case‑fatality among encephalitis cases can be 10–30% depending on setting.
  • Of survivors, 30–50% may have long‑term sequelae (motor deficits, seizures, cognitive/behavioral changes).

Prevention

  • Vaccination: highly effective for residents/travelers at risk; follow local product schedules (e.g., inactivated Vero‑cell vaccines or live‑attenuated SA14‑14‑2).
  • Mosquito avoidance: repellents (DEET/picaridin/IR3535), long sleeves/pants, permethrin‑treated clothing/bed nets, screened housing; reduce standing water.

Red Flags — Seek Urgent Care

  • High fever with severe headache/neck stiffness, seizures, confusion/coma, new weakness, breathing difficulty.

Educational information only; suspected JE warrants urgent hospital care and public‑health notification.

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